Miscellanous Tutorial
DRESS syndrome
DRESS syndrome is associated with a morbiliform skin rash.
Micronutrient deficiency
Angular cheilitis
Scaling and erythema of the oral commissures, associated with a number of nutritional deficiencies.
Acrodermatitis
Scaly erythematous rash around the anogenital area, usually associated with zinc deficiency.
Glossitis
Red beefy tongue.
Scurvy
Reflex sympathetic dystrophy
Poorly understood cause of pain in a region of the body, usually starting in a limb after trauma, but the pain is disproportionate to the severity of the original injury
Characterized by pain, swelling, and characteristic changes to the skin, such as reddening with a waxy or shiny appearance.
Dermatitis
Dermatitis and eczema are terms that denote an inflammatory reaction involving the epidermis and dermis, and are used interchangeably. There are many forms of dermatitis.
- Atopic dermatitis–The result of a complex interaction between genetic and environmental factors. Usually manifests as dry, itchy skin. Poorly defined erythematous patches, papules, and plaques, sometimes with scale.
- Allergic contact dermatitis–Caused by an antigen that triggers a type IV (cell-mediated) hypersensitivity reaction.It usually manifests as erythema and edema with superimposed papules or vesicles that are sharply demarcated, corresponding to the areas of exposure.
- Irritant contact dermatitis–Caused by a chemical irritant, usually occurring minutes or up to 24 hours after exposure. It usually manifests as erythema and/or vesiculation that are sharply demarcated, corresponding to the areas of exposure.
- Stasis dermatitis-Chronic venous insufficiency occurs when dysfunction of the venous system of the lower extremitiesleads to venous hypertension. This process most commonly develops as a result of venous valvular incompetence related to previous clinical or occult DVTs. Clinical sequelae include pain, edema, and skin changes such as telangiectasias, varicosities, hemosiderosis-related hyperpigmentation, lipodermatosclerosis (a fibrotic process involving the dermis and subcutaneous fat), and ulcerations (typically near the medial malleoli).